Autre Nora KIRSTEN, Autre M.N. KALFF, Autre L.A PARDO Jr, Autre K KASPRZAK, Autre M. EGGER, Autre V. WITOWSKI, Autre S.N. SCHMIDT, Autre S. SEHMISCH, Autre J. ERNST
The Agonist-Antagonist Myoneural Interface (AMI) represents an innovative approach to restoring proprioception and achieving more intuitive motor control following limb loss. This cutting-edge technique replicates the natural biomechanical relationship between agonist and antagonist muscles, enabling bidirectional communication between a prosthesis and the user's peripheral nervous system.
Agonist-Antagonist Myoneural Interface (AMI)
Through the transposition of neurovascularly pedicled agonist-antagonist muscle pairs, which are re-connected via an adapted tendon suture and positioned within a gliding mechanism- AMI generates proprioceptive feedback during movement. Changes in tension within these muscle pairs produce signals that are transmitted to the central nervous system via afferent nerve pathways, allowing users to perceive the joint position of the limb that was originally governed by the muscle pair. This enhanced sensory input significantly facilitates prosthetic control.
AMI integrates the prosthesis more seamlessly into the body's existing neural networks, offering improved motor control and heightened sensory discrimination compared to traditional surgical techniques, which often rely on a purely mechanical transposition of residual stump muscles. Unlike conventional approaches such as myodesis or myoplasty, AMI reduces the cognitive burden of prosthetic use and delivers a more natural sense of movement, fostering a profound sense of embodiment.
In summary, AMI represents a significant leap forward in human-machine integration. By enhancing both the functionality and user experience of prosthetic devices, it offers a transformative solution for improving the quality of life for individuals living with limb loss.
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